For example, human SET7/9 has just lately been reported to methylate Lys189 in the normal transcription factor TAF10, resulting in an elevated affinity for RNA polymerase II and transcriptional activation of sure TAF10-dependent genes 37 SET7/9 has additionally been reported to methylate p53, increasing the stability of this brief lived tumor-suppressor protein 38 These observations recommend that we should always not narrowly define the SET-domain proteins as histone lysine methyltransferases but instead call them protein lysine methyltransferases.

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